鎂離子（蘇氨酸鎂Magnesium Threoninate、蘋(píng)果酸鎂Magnesium Malate、抗壞血酸鎂Magnesium Ascorbate）在運(yùn)動(dòng)損傷中的作用

Mg元素可解除平滑肌痙攣，使血管擴(kuò)張，Mg²⁺對(duì)鈣離子在肌質(zhì)網(wǎng)膜的轉(zhuǎn)運(yùn)起著重要作用，會(huì)影響一些陰離子與肌質(zhì)網(wǎng)膜的結(jié)合，對(duì)骨折后的治療起重要作用。在人體內(nèi)的中樞神經(jīng)，周?chē)窠?jīng)，肌肉及心血管系統(tǒng)方面，Mg²⁺的作用更是顯而易見(jiàn)，因Mg²⁺具有類(lèi)似箭毒作用，能抑制神經(jīng)肌肉接頭及乙酞膽堿的釋放和運(yùn)動(dòng)終板對(duì)乙酞膽堿的敏感性。Mg²⁺對(duì)中樞神經(jīng)也有抑制作用，在血漿中鎂達(dá)10毫克當(dāng)量每升時(shí)，就有催眠及麻痹作用。深鍵反射消失，若濃度更高，可引起呼吸中樞抑制，在心血管系統(tǒng)方面， Mg²⁺直接抑制房室和心室內(nèi)傳導(dǎo)，并降低心肌的應(yīng)激性，經(jīng)生理實(shí)驗(yàn)證明，當(dāng)血漿鎂在5-10毫克當(dāng)克/升時(shí)則出現(xiàn)心動(dòng)過(guò)緩及傳導(dǎo)阻滯，若濃度達(dá)22-44毫克當(dāng)量1升時(shí)，則心肌停搏，同時(shí)鎂還使外周小動(dòng)脈松馳，血壓下降等。

一般成年人每日對(duì)鎂的需求量，男性為350毫克，女性為300毫克， 如果缺鎂，可導(dǎo)致肌肉無(wú)力，耐久力降低。由于運(yùn)動(dòng)，特別是長(zhǎng)時(shí)間高強(qiáng)度運(yùn)動(dòng)大量消耗體內(nèi)的鎂，從而降低肌肉的活動(dòng)功能，甚至還會(huì)發(fā)生抽搐、痙攣，容易出現(xiàn)不同程度和部位的軟組織損傷。所以，為避免由于運(yùn)動(dòng)而引起肌肉損傷, 應(yīng)補(bǔ)充一定的鎂（蘇氨酸鎂Magnesium Threoninate、蘋(píng)果酸鎂Magnesium Malate、抗壞血酸鎂Magnesium Ascorbate）。

（蘇氨酸鎂Magnesium Threoninate、蘋(píng)果酸鎂Magnesium Malate、抗壞血酸鎂Magnesium Ascorbate）在運(yùn)動(dòng)損傷中的作用

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抗壞血酸鎂Magnesium Ascorbate 、抗壞血酸鋅Magnesium Ascorbate、牛磺酸鎂Magnesium Taurate、氨基酸螯合鋅Zinc Amino Acids Chelate 、氨基酸螯合鎂Magnesium Amino Acids Chelate、乳清酸鎂Magnesium Orotate、乳清酸鈣Calcium Orotate、琥珀酸鈣Calcium Succinate、琥珀酸鎂 Magnesium Succinate 、抗壞血酸鋰 Lithium Ascorbate.

The role of magnesium ions (Magnesium Threoninate, Magnesium Malate, Magnesium Ascorbate) in sports injuries

Mg can relieve smooth muscle spasms and dilate blood vessels. Mg²⁺ plays an important role in the transport of calcium ions in the sarcoplasmic reticulum, affects the binding of some anions to the sarcoplasmic reticulum, and plays an important role in the treatment of fractures. In the central nervous system, peripheral nerves, muscles and cardiovascular systems in the human body, the role of Mg²⁺ is even more obvious, because Mg²⁺ has a similar effect to curare, which can inhibit the release of neuromuscular junctions and acetylcholine and the sensitivity of motor end plates to acetylcholine. Mg²⁺ also has an inhibitory effect on the central nervous system. When the magnesium content in plasma reaches 10 milligrams per liter, it has a hypnotic and paralytic effect. The deep key reflex disappears. If the concentration is higher, it can cause respiratory center inhibition. In terms of the cardiovascular system, Mg²⁺ directly inhibits atrioventricular and intraventricular conduction and reduces myocardial irritability. Physiological experiments have shown that when plasma magnesium is 5-10 mg/L, bradycardia and conduction block occur. If the concentration reaches 22-44 mg/L, myocardial arrest occurs. At the same time, magnesium also relaxes peripheral arterioles and reduces blood pressure.

The daily magnesium requirement for adults is 350 mg for men and 300 mg for women. If magnesium is deficient, it can lead to muscle weakness and reduced endurance. Exercise, especially long-term high-intensity exercise, consumes a lot of magnesium in the body, thereby reducing muscle activity function, and even convulsions and spasms may occur, which is prone to soft tissue injuries of varying degrees and locations. Therefore, in order to avoid muscle damage caused by exercise, a certain amount of magnesium should be supplemented (magnesium threoninate, magnesium malate, and magnesium ascorbate).

(Magnesium Threoninate, Magnesium Malate, Magnesium Ascorbate) in sports injuries

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Magnesium Ascorbate, Magnesium Ascorbate, Magnesium Taurate, Zinc Amino Acids Chelate, Magnesium Amino Acids Chelate, Magnesium Orotate, Calcium Orotate, Calcium Succinate, Magnesium Succinate, Lithium Ascorbate.