鋅元素（甘氨酸鋅Zinc Glycinate、L-天門冬氨酸鋅Zinc Aspartate 、乳清酸鋅Zinc Orotate、抗壞血酸鋅Zinc Ascorbate、葡萄糖酸鋅Zinc Gluconate、檸檬酸鋅Zinc Citrate、蘋果酸鋅Zinc Malate）對(duì)人體的影響

鋅是生長(zhǎng)和發(fā)育的必須元素。氨基酸、核酸、脂肪、碳水化合物及微量元素等營(yíng)養(yǎng)元素的代謝均需要鋅的參與，而且胰島素、胰高血糖素、前列腺素和促性腺激素等的活動(dòng)也與鋅有關(guān)。在細(xì)胞水平上，它與細(xì)胞的增值、分化和凋亡有密切的關(guān)系。鋅的其他功能包括免疫、中間代謝、DNA代謝、修復(fù)和再生、視力、味覺、認(rèn)識(shí)能力或行為。另外，鋅也是神經(jīng)形成、突觸發(fā)生、神經(jīng)元生長(zhǎng)和神經(jīng)傳遞的必需元素。它存儲(chǔ)在特殊的谷氨酰胺神經(jīng)元突觸小泡中并以神經(jīng)調(diào)節(jié)物的形式釋放。缺鋅會(huì)發(fā)生繼發(fā)疾病損害腸道吸收或提高腸道鋅的損失，例如，腸病性肢端皮炎；也會(huì)引起動(dòng)物機(jī)體物質(zhì)代謝紊亂和生長(zhǎng)發(fā)育受阻，機(jī)體的免疫功能的降低，繁殖機(jī)能受到損傷。在兒童和青少年中，發(fā)育不良和發(fā)育遲緩可能是最早被發(fā)現(xiàn)的缺鋅的標(biāo)志。缺鋅患者除可表現(xiàn)為傷口愈合緩慢外并無其他顯著癥狀，頭發(fā)容易變的粗細(xì)不均，明顯脫發(fā)，黑色的頭發(fā)會(huì)變成紅棕色。對(duì)神經(jīng)系統(tǒng)的影響可能包括降低神經(jīng)傳導(dǎo)，共濟(jì)失調(diào)，神志不清，和神經(jīng)心理受損的表現(xiàn)。缺鋅初期表現(xiàn)并不明顯，缺鋅已經(jīng)存在了一段時(shí)間之后，其他的表現(xiàn)可能會(huì)出現(xiàn)，包括生殖器的發(fā)育遲緩和性腺功能低下，妊娠不良結(jié)局和畸形兒，痢疾、肺炎和其他傳染病的發(fā)病和和大腦功能受損。

鋅元素（甘氨酸鋅Zinc Glycinate、L-天門冬氨酸鋅Zinc Aspartate 、乳清酸鋅Zinc Orotate、抗壞血酸鋅Zinc Ascorbate、葡萄糖酸鋅Zinc Gluconate、檸檬酸鋅Zinc Citrate、蘋果酸鋅Zinc Malate）對(duì)人體的影響

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抗壞血酸鎂Magnesium Ascorbate、抗壞血酸鈣Calcium Ascorbate、甘氨酸鎂Magnesium Glycinate、甘氨酸谷氨酰胺鎂Magnesium Glycinate Glutamine、檸檬酸蘋果酸鈣Calcium Citrate Malate、蘋果酸鎂Magnesium Malate、檸檬酸錳Manganese Citrate、檸檬酸鎂Magnesium Citrate、天門冬氨酸鈣Calcium Aspartate、牛磺酸鎂Magnesium Taurate.

Zinc elements (Zinc Glycinate, Zinc Aspartate, Zinc Orotate, Zinc Ascorbate, Zinc Gluconate, Zinc Citrate、Zinc Malate) on the human body

Zinc is essential for growth and development. The metabolism of amino acids, nucleic acids, fats, carbohydrates and trace elements requires the participation of zinc, and the activities of insulin, glucagon, prostaglandin and gonadotropin are also related to zinc. At the cellular level, it is closely related to cell proliferation, differentiation and apoptosis. Other functions of zinc include immunity, intermediate metabolism, DNA metabolism, repair and regeneration, vision, taste, cognition, or behavior. In addition, zinc is essential for neurogenesis, synaptogenesis, neuron growth, and neurotransmission. It is stored in special synaptic vesicles of glutamine neurons and released as a neuromodulator. Zinc deficiency can result in secondary diseases that impair intestinal absorption or increase intestinal zinc loss, such as enteropathic acrodermatitis; It can also cause disorder of animal body material metabolism and growth and development, the body's immune function is reduced, and the reproductive function is damaged. In children and adolescents, stunting and stunting may be the earliest markers of zinc deficiency to be identified. In addition to slow wound healing, zinc deficiency patients have no other significant symptoms, hair is easy to become uneven thickness, obvious hair loss, black hair will turn reddish brown. Effects on the nervous system may include reduced nerve conduction, ataxia, confusion, and neuropsychological impaired manifestations. Initial symptoms of zinc deficiency are not obvious, and after zinc deficiency has been present for some time, other symptoms may appear, including genital stunting and hypogonadism, adverse pregnancy outcomes and birth defects, increased morbidity and mortality from diarrhea, pneumonia and other infectious diseases, and impaired brain function.  

Zinc elements (Zinc Glycinate, Zinc Aspartate, Zinc Orotate, Zinc Ascorbate, Zinc Gluconate, Zinc Citrate、Zinc Malate) on the human body

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Magnesium Ascorbate、Calcium Ascorbate、Magnesium Glycinate、Magnesium Glycinate Glutamine、Calcium Citrate Malate、Magnesium Malate、Manganese Citrate、Magnesium Citrate、Calcium Aspartate、Magnesium Taurate.